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. 2013 Sep 15;27(18):1949–1958. doi: 10.1101/gad.220194.113

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© 2013, Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genesdev.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 3.0 Unported), as described at http://creativecommons.org/licenses/by-nc/3.0/.

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Figure 2. — Development of mGS cells after Dmrt1 knockdown (KD). (A) Real-time PCR analysis of GCT candidate gene expression in p53 knockout (KO) GS cells after Dnmt1 knockdown (n = 3). pSicoR was used as a control. (B) Suppression of mGS cell development by Dmrt1 overexpression (OE) 28 d after Dnmt1 knockdown (n = 9). CSII-EF1α-IRES2-Venus was used as a control. (C) Suppression of Dmrt1-induced apoptosis by p53 or Bax knockdown. For each cell type, at least 68 cells were counted 7 d after transfection (n = 5).