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editorial

. 2013 Oct 11;4:401. doi: 10.3389/fpls.2013.00401

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Copyright © 2013 Serna.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Model for the regulation of the self-renewal behavior of the meristemoids and their transition to guard mother cell (GMC) fate. Predominance of SPCH, which is activated by YDA MAPK module, inhibits the meristemoid-to-GMC transition by promoting the self-renewal behavior of the meristemoids. In contrast, predominance of MUTE represses the amplifying divisions of the meristemoids and enhances their transition through the GMC fate. GMC symmetrically divides to produce paired-guard cells (not shown). The components upstream of MUTE remain unknown. SCRM1 and SCRM2 function in this transition through direct interaction with SPCH and MUTE (not shown).