Skip to main content
. Author manuscript; available in PMC: 2014 Oct 3.
Published in final edited form as: Int J Cardiol. 2013 Apr 15;168(3):2567–2574. doi: 10.1016/j.ijcard.2013.03.049

Fig. 1.

Fig. 1

G-CSF effects on myocardial inflammatory lesions and parasite burden in T. cruzi-infected mice. C3H/He mice were infected with T. cruzi and treated with G-CSF at day 21 or 1 year post-infection. Mice were harvested after the completion of treatment. (A) Microphotographs of tissue-sections from acutely and chronically infected mice (±G-CSF) stained with H&E and examined for inflammation and intracellular parasites (black arrows, magnification: 200×; bar: 100 μm). (B) T. cruzi 18SrDNA sequence amplification by PCR (25 cycles) in heart tissue from infected mice. Murine GAPDH was amplified as a control. NI: uninfected control, I: infected, I + GCSF: infected/G-CSF-treated.