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. Author manuscript; available in PMC: 2013 Oct 22.
Published in final edited form as: Psychiatry Res. 2009 May 14;173(1):63–70. doi: 10.1016/j.pscychresns.2008.09.004

Table 1.

FDG-PET studies of treatment outcome in major depression: table summarizes the 18F-FDG-PET identified in the literature past 1996 that reported on regional brain glucose metabolic rate as a biological predictor of treatment outcome.

Authors and year N Treatment Imaging analysis Brain region
Mesiotemporal
cortex
Dorsal prefrontal
cortex
Ventral prefrontal
cortex
Anterior cingulate
Posterior
cingulate
Insula
Parietal
Temporal
Occipital
Basal ganglia
Thalamus
Cerebellum
Midbrain
L/R/B
L/R/B
L/R/B
L/R/B
L/R/B
L/R/B
L/R/B
L/R/B
L/R/B
L/R/B
L/R/B
L/R/B
L/R/B
Rsp NR Rsp NR Rsp NR Rsp NR Rsp NR Rsp NR Rsp NR Rsp NR Rsp NR Rsp NR Rsp NR Rsp NR Rsp NR
Little et al. (1996) 11 Venlafaxine or buproprion
(randomized, with cross-over)
SPM –/–/↓° –/–/↓° –/–/↓°
Buchsbaum etal. (1997) 17 Sertraline (placebo-controlled,
randomized)
BMP/correlation ↑/–/–
Mayberg (1997) 18 SSRI, TCA, buproprion ROI –/↑/– –/↓/–
(non-randomized) SPM ↓/v/–C –/–/↓ –/↑/– –/↓/– –/↓/–
Brody etal. (1999) 16 Paroxetine ROI/correlation ↓/–/–
Wu et al. (1999) 36 Sleep deprivation ROI –/–/↑ –/↑/– –/↓/– –/↓/– –/↓↑/–D –/↓/–D ↑/–/– –/↓/– ↓/–/– –/↓/–
*ROI –/↓/– ↓/–/– –/↑/↑A –/–/↑ –/↓/–
Brannan et al. (2000) 53 Antidepressants ↑/–/– –/↑/– –/–/↑
Brody et al. (2001) 24 Paroxetine or ITP ROI –/↑/– ↑/–/–
(non-randomized) SPM –/–/↑ –/–/↑ –/↑/–
Kennedy et al. (2001) 13 Paroxetine SPM ↓/–/–phc –/↑/– –/↓/–
Saxena et al. (2003) 27 Paroxetine ROI/correlation –/↓/–
44B SPM –/–/↑ –/↑/–
Little et al. (2005) 20 Venlafaxine or buproprion SPM ↓/–/– –/–/↓ –/–/↓ –/–/↑ /–/↓E –/↓/–E –/↓/– ↓/–/–F –/–/↓C –/–/↑G ↑/–/–G –/↓/↓° –/–/↓C c –/–/↑
(randomized, with cross-over) SPM (absolute) ↑/–/–
*SPM ↓/–/– ↓/–/– –/–/↓ ↑/–/–F ↓/–/–
*SPM (absolute) ↓/–/– ↓/–/– –/–/↓

Abbreviations: Comparison group are Controls unless otherwise indicated; *Comparison of Responders to Non-responders; N=number of depressed subjects, L/R/B=Left, Right, Bilateral, BMP=Biomedical Computer Program, SPM=Statistical Parametric Mapping, All SPM data are from normalized rCMRglu unless otherwise indicated. Results of Little et al. (1996) are from both absolute and normalized rCMRglu, A=data from 21 subjects not studied previously, B=MDD +/− comorbid OCD, C=decrease rCMRglu in right Brodmann area 44 and left Brodmann area 46/49 are unique to non-responders, phc=post hoc analysis, D=non-responders express decrease rCMRglu in Brodmann area 38/21, increase rCMRglu is seen in responders, decrease rCMRglu in responders is located in Brodmann area 37, E=non-responders to buproprion demonstrated decrease rCMRglu in right Brodmann area 32, while responders to venlafaxine demonstrated decrease rCMRglu in Brodmann area 32 bilaterally, F=non-responders to buproprion demonstrated decreased rCMRglu in left Brodmann area 4, responders to either medication demonstrated increase rCMRglu in left Brodmann area 40, G=venlafaxine non-responders only, buproprion responders only; ITP=Interpersonal therapy, °=coordinates not reported from SPM analysis.