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. 2013 Oct 23;8(10):e74123. doi: 10.1371/journal.pone.0074123

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© 2013 Tovar-Lopez et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Top stream (85): labelled whole blood. Bottom stream (15): Autologus platelet-poor-plasma (PPP). a) Representative DIC images of blood perfusion experiments over 10 minutes of monitoring in a device that produces two non-symmetrically focused streams ( and ). It can be observed that contrary to previous experiments, no aggregate was formed downstream of the contraction. b) Representative (n = 3) epi-fluorescence images of the same experiment as (a)). No aggregate was formed downstream of the contraction. This data demonstrates that initial aggregate formation was driven by platelets skimming within of the stenosis apex.

Inline graphic — Top stream (85): labelled whole blood. Bottom stream (15): Autologus platelet-poor-plasma (PPP). a) Representative DIC images of blood perfusion experiments over 10 minutes of monitoring in a device that produces two non-symmetrically focused streams ( and ). It can be observed that contrary to previous experiments, no aggregate was formed downstream of the contraction. b) Representative (n = 3) epi-fluorescence images of the same experiment as (a)). No aggregate was formed downstream of the contraction. This data demonstrates that initial aggregate formation was driven by platelets skimming within of the stenosis apex.