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. Author manuscript; available in PMC: 2014 Jul 9.

Published in final edited form as: Circulation. 2013 Jun 11;128(2):10.1161/CIRCULATIONAHA.112.001075. doi: 10.1161/CIRCULATIONAHA.112.001075

Assessment of LV function before and after vehicle or CSC therapy: hemodynamic variables [LV end-diastolic pressure (A) and LV dP/dt_max (B)], representative M-mode echocardiographic images at 30 d after treatment in pigs that were given vehicle (C) and CSCs (D), and quantitative echocardiographic analysis of LV function (IW thickening fraction and infarcted wall thickening fraction) (E and F). Compared with the vehicle-treated pig, the CSC-treated animal exhibited a smaller LV cavity, a thicker infarcted wall, and improved motion of the infarcted wall (C and D), Quantitative echocardiographic analysis shows improvement in LV functional parameters at 30 d after CSC treatment (E and F). Data are means ± SEM. *, P<0.05 versus noninfarcted controls and §, P<0.05 vs. vehicle-treated pigs (unpaired t test).

Assessment of LV function before and after vehicle or CSC therapy: hemodynamic variables [LV end-diastolic pressure (A) and LV dP/dt_max (B)], representative M-mode echocardiographic images at 30 d after treatment in pigs that were given vehicle (C) and CSCs (D), and quantitative echocardiographic analysis of LV function (IW thickening fraction and infarcted wall thickening fraction) (E and F). Compared with the vehicle-treated pig, the CSC-treated animal exhibited a smaller LV cavity, a thicker infarcted wall, and improved motion of the infarcted wall (C and D), Quantitative echocardiographic analysis shows improvement in LV functional parameters at 30 d after CSC treatment (E and F). Data are means ± SEM. *, P<0.05 versus noninfarcted controls and §, P<0.05 vs. vehicle-treated pigs (unpaired t test).