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. 2013 Sep 24;99(22):1663–1667. doi: 10.1136/heartjnl-2013-304624

Table 2.

Outcomes and adverse events

N=19
Suspected infarct localisation, no./total no. (%)
 Anterior wall infarction 9/19 (47%)
 Posterior wall infarction 10/19 (53%)
Pre-PCI TIMI grade flow*, no./total no. (%)
 0 13/19 (68%)
 1 3/19 (16%)
 2 0
 3 3/19 (16%)
Culprit lesion, no./total no. (%)
 No significant coronary stenosis 3/19 (16%)
 Left main artery 1/19 (5%)
 Left anterior descending artery 9/19 (47%)
 Circumflex artery 2/19 (11%)
 Right coronary artery 4/19 (21%)
Post-PCI TIMI grade flow*, no./total no. (%)
 0 1/19 (5%)
 1 0
 2 1/19 (5%)
 3 17/19 (90%)
Peak level of creatine kinase, units/L (IQR)† 3807 (1429–5585)
Peak level of troponin T, ng/mL (IQR)† 4.56 (2.50–10.00)
Killip classification, no./total no. (%)
 I 13/19 (68%)
 II 3/19 (16%)
 III 2/19 (11%)
 IV 1/19 (5%)
Recurrent ischaemia, no./total no. (%) 0
Major cardiac arrhythmias, no./total no. (%)
 Ventricular tachycardia 2/19 (11%)
 Ventricular fibrillation 0
 Asystole 0
 AV-Block ≥2° 0
Stroke 0
Major/minor bleeding§ 0
Cardiac arrest 1/19 (5%)
Death 1/19 (5%)
NYHA class at day 30§
 I 10/18 (56%)
 II 6/18 (33%)
 III 2/18 (11%)
 IV 0
Infection within 30 days, no./total no. (%) 3/19 (16%)

*TIMI grade flow prior/after percutaneous coronary intervention.

†of 15 patients with percutaneous coronary intervention.

‡according to thrombolysis in myocardial infarction bleeding score.

§NYHA category of 18 patients survived to follow-up.

NYHA, New York Heat Association; PCI, percutaneous coronary intervention; TIMI, thrombolysis in myocardial infarction.