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. 2013 Nov;195(3):883–898. doi: 10.1534/genetics.113.156984

Table 2. Fusion phenotype of strains carrying deletions that showed significant differences in expression level in the Δpp-1 mutant relative to wild type.

NCU ID no. WT RPKM Δpp-1 RPKM Protein description Deletion origin Fusion % (WT = 86 ± 4)a
NCU00309b 333 527 Hypothetical FGSC 19692 WT
NCU00811 1302 630 Hypothetical This study WT
NCU00881 145 67 HAM7 13775 0
NCU00995 274 0 Hypothetical This study WT
NCU01380 413 1 Hypothetical This study WT
NCU01697 130 2 Hypothetical FGSC 14575 WT
NCU02361 42 117 Formamidase FGSC 21969 WT
NCU02500 49 642 CCG4 FGSC 14944 WT
NCU03013 368 25 ACW10 FGSC 11222 WT
NCU03960 200 24 HAM12 FGSC 17233 66 ± 6
NCU04122 269 19 Malate dehydrogenase FGSC 21282 WT
NCU04192 70 42 Vacuolar aspartyl aminopeptidase FGSC 18884 WT
NCU04732 179 65 HAM11 FGSC 17545 and this study 0
NCU05814 145 48 Hypothetical FGSC 17770 WT
NCU06698 79 49 Glycogenin FGSC 12301 WT
NCU07503 77 300 Hypothetical FGSC 14018 WT
NCU07802 148 1 Hypothetical FGSC 20487 WT
NCU08332 1422 448 HEX1 Jedd and Chua (2000) WT
NCU08824 37 73 Molybdopterin-binding domain FGSC 15980 WT
NCU09560 66 150 Superoxide dismutase FGSC 21068 WT
NCU09562 501 5 Hypothetical FGSC 21874 WT
NCU09693 116 5 Hypothetical This study WT
a

Mutants show fusion frequencies similar to their isogenic wild-type parent (FGSC 2489).

b

Genes in boldface type showed increased expression levels in the Δpp-1 mutant.