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. 2013 Nov 5;8(11):e78535. doi: 10.1371/journal.pone.0078535

Table 1. Behavior of sensory and pheromone mutants.

Genotype Gene function Sensory defect Directionality p-value
che-2 cilia formation chemotaxis (‘ctx’) defective 0.003 (vs. WT)
daf-19* cilia formation (master regulator) chemotaxis (‘ctx’) partially defective 0.03 (vs. WT)
osm-9 sensory transduction (TRPV-related channel) avoidance of repulsive compounds; nociception; ctx to some volatile compounds wild-type 0.0008 (vs. CRW)
tax-4(p678) sensory transduction (cGMP-gated channel) thermotaxis; aerotaxis; ctx to some volatile and soluble compounds. defective 0.008 (vs. WT)
tax-4(ks28) sensory transduction (cGMP-gated channel) same as tax-4(p678) defective 0.006 (vs. WT)
tax-2 sensory transduction (cGMP-gated channel) same as tax-4(p678) defective 0.009 (vs. WT)
gcy-8;gcy-18;gcy-23 sensory transduction (guanylyl cyclases) thermotaxis wild-type 0.0003 (vs. CRW)
gcy-31;gcy-33;gcy-35 sensory transduction (guanylyl cyclases) aerotaxis wild-type 0.003 (vs. CRW)
gcy-36::EGL-1 sensory neurons ablated aerotaxis wild-type 0.0002 (vs. CRW)
odr-1 sensory transduction (guanylyl cyclase) ctx to volatile compounds sensed by TAX-4 neurons; phototactic avoidance wild-type 0.004 (vs. CRW)
che-1 sensory transduction (specification of sensory neuronal fate) ctx to soluble compounds wild-type 0.02 (vs. CRW)
daf-22 C. elegans pheromone synthesis dauer formation wild-type 0.01 (vs. CRW)

Significance is estimated with respect to the wild-type scanner-array data-set (‘WT’) or to the correlated random walk model (‘CRW’). *daf-19 mutants were assayed in a daf-16 background to bypass constitutive dauer formation.