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. 2013 Oct 14;14(10):20282–20298. doi: 10.3390/ijms141020282

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© 2013 by the authors; licensee MDPI, Basel, Switzerland

This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution license (http://creativecommons.org/licenses/by/3.0/).

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Effects of Rho kinase inhibition on oxidative stress in intrarenal artery and EDCFs in plasma from diabetic rats. (A) The concentrations of TxB₂ and PGF_2α in plasma of rats after 4-week treatment with fasudil. (B) Fluorescent photomicrographs at identical settings of sections of intrarenal artery labeled with dihydroethidium and quantification of the fluorescent ethidium signal in the artery by average intensity. Scale bar is 50 μm shown in the picture. *p < 0.05, ***p < 0.001 compared with control group; ^##p < 0.01 when compared diabetic rats with and without treatment of fasudil. Data are mean ± S.E.M. from four–six different animals.

Effects of Rho kinase inhibition on oxidative stress in intrarenal artery and EDCFs in plasma from diabetic rats. (A) The concentrations of TxB₂ and PGF_2α in plasma of rats after 4-week treatment with fasudil. (B) Fluorescent photomicrographs at identical settings of sections of intrarenal artery labeled with dihydroethidium and quantification of the fluorescent ethidium signal in the artery by average intensity. Scale bar is 50 μm shown in the picture. *p < 0.05, ***p < 0.001 compared with control group; ^##p < 0.01 when compared diabetic rats with and without treatment of fasudil. Data are mean ± S.E.M. from four–six different animals.