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. 2013 Nov 12;7:179. doi: 10.3389/fncir.2013.00179

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Copyright © 2013 Viemari, Garcia, Doi, Elsen and Ramirez.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Activation of β-NR modulates fictive sigh. (A) ∫VRG activity recorded from a slice under control conditions. (B) Isoproterenol 20 μM (β-NR agonist) activates β-NR and increases specifically the frequency of sigh activity. (C–F) Histograms show the effects of isoproterenol on sigh burst frequency (E) without affecting respiratory activity (C), sigh burst amplitude (D), or the sigh burst area (F) (^*p < 0.05, n = 10).