Table 3.

Demographics, neurological examination, MRI, mf density and skin PCR of group 2

Age	Gender	Seizure	Neurological examination/mental	Intraparenchymal	Atrophy	Mf skin/mg	PCR
(yrs)		type	examination	hymal MRI pathologies			skin
14	F	1.Gen+, 2.Cp	NAD	No	No	0.5	Not tested
16	M	Gen	MeRe, no focal neurological signs	No	Cerebellar vermis	4	Neg
18	F	Gen	MeRe, no focal neurological signs	Arachnoid cyst rt frontal	No	35.5	Pos
22	M	Gen+	MeRe, hemihypaesthesia rt, UMN signs rt	Susp. HC sclerosis bilateral	Mild general	0.17	Pos
47	M	1.Gen, no focal neurological signs	NAD	HC sclerosis lft, susp. HC sclerosis rt; signal abnorm. subcortial bilateral frontal	Cerebellar	5.5	Pos
13	F	Cp	MeRe, no focal neurological signs	No	No	0	Pos
13	M	1.Gen, 2.Cp	MeRe, no focal neurological signs	Signal abnorm. subcortical bilateral occipital	No	0	Neg
15	M	Gen	MeRe, cerebellar syndrome	Cystic defects occipital bilateral	Cerebellar	0	Neg
16	M	Gen	MeRe, increased reflexes	No	Mild cerebellar	0	Neg
27	F	Gen+	NAD	Susp. HC sclerosis bilateral	Cerebral occipital + parietal	0	Neg

Cp - complex partial seizures, Gen - generalized tonic clonic seizures, Gen+ - generalized tonic clonic seizures with additional focal signs ictally, NAD - nothing abnormal detected, MeRe - mental retardation, UMN - upper motor neuron, HC - Hippocampus, lft - left, rt - right, susp. - suspected, abnorm. - abnormalities, Mf - microfilariae, HC changes were divided into HC sclerosis and suspected HC sclerosis, the latter only showing either HC signal changes or volume loss.