Table 4.

Demographics, neurological examination, MRI, mf density and skin PCR of group 3

Age	Gender	Seizure	Neurological examination/mental	Intraparenchymal	Atrophy	Mf	PCR skin
(yrs)		type	examination	MRI pathologies		skin /mg
15	M	HN only	NAD	No	No	1.5	Neg
16	F	HN only	NAD	Gliotic lesions rt frontal	No	5.65	Pos
				subcortical; susp. HC
				sclerosis rt>lft
18	M	HN only	Frontal release signs,	No	No	1	Neg
			UMN signs lft>rt
10	F	HN only	NAD	No	No	0	Not tested
14	F	HN only	NAD	No	No	Neg*	Not tested
15	M	HN only	NAD	Arachn cyst rt	No	0	Pos
				temporal; HC sclerosis rt
9	F	HN only	UMN signs rt	Gliotic lesions rt frontal	General	Pos*	Not tested
				subcortical; susp. HC
				sclerosis rt>lft
15	M	1.HN,	NAD	Gliotic lesions bilateral	No	1.5	Pos
		2.Gen		lft>rt frontal subcortical
16	F	1.HN, 2.Gen	NAD	Gliotic lesions lft temporal-occipital-parietal gwm-j	No	6.17	Pos
13	M	1.HN,	NAD	Gliotic lesions bilateral	No	0	Neg
		2.Gen		frontal subcortical
16	F	1.HN,	NAD	Susp. HC sclerosis lft	0	Pos
		2.Gen		Mild cerebellar
21	M	1.HN,	Brain damage, no focal	Susp. HC sclerosis	Mild	0	Pos
		2.Gen,	neurology	bilateral	cerebellar
		3.Cp

HN - head nodding seizures, Gen - generalized tonic clonic seizures, Cp - complex partial seizures, NAD - nothing abnormal detected, HC - Hippocampus, susp. - suspected, gwm-j - grey-white matter junction, rt - right, lft - left, neg*, pos* - no further counting available

HC changes were divided into HC sclerosis and suspected HC sclerosis, the latter only showing either HC signal changes or volume loss.