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Expression of anti-apoptotic Bcl-2 family proteins or their phospho-defective mutants inhibits caspase-3 activation in response to vinblastine. Control HeLa cells or cells stably overexpressing the indicated Bcl-2 family protein were untreated or treated with 30 nM vinblastine for 24 or 48 h, and cell extracts were subjected to caspase-3 assay, as described in Materials and Methods. Results given are mean±S.D. (n=3)
