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. 2013 Nov 1;27(21):2345–2355. doi: 10.1101/gad.223800.113

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Published by Cold Spring Harbor Laboratory Press

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Figure 6. — Thyroid hormone is required for glycemic regulation caused by JNK deficiency in the pituitary gland. (A) The total body mass gain of PTU-treated ND-fed and HFD-fed mice treated with PTU in the drinking water was examined (mean ± SEM; n = 7∼10). No significant differences between P^WT and P^ΔJ1,J2 mice were detected. P > 0.05. (B) The fat and lean mass of PTU-treated ND-fed and HFD-fed (16 wk) mice were measured by ¹H-MRS analysis (mean ± SD; n = 7∼10). No significant differences between P^WT and P^ΔJ1,J2 mice were detected. P > 0.05. (C) The effect of consuming a HFD (16 wk) on glucose tolerance of PTU-treated mice was examined (mean ± SEM; n = 8∼12). No significant differences between P^WT and P^ΔJ1,J2 mice were detected. P > 0.05. (D) PTU-treated P^WT and P^ΔJ1,J2 mice were fed an ND or a HFD (16 wk). The blood glucose concentration in mice fasted overnight or fed ad libitum was measured (mean ± SEM; n = 7∼12). P > 0.05.