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. 2013 Aug 23;2(4):e000260. doi: 10.1161/JAHA.113.000260

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© 2013 The Authors. Published on behalf of the American Heart Association, Inc., by Wiley-Blackwell.

This is an Open Access article under the terms of the Creative Commons Attribution Noncommercial License, which permits use, distribution, and reproduction in any medium, provided the original work is properly cited and is not used for commercial purposes.

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Figure 2. — Genomewide QTL analysis to search for loci influencing atherosclerotic lesion sizes in male F₂ mice. Chromosomes 1 through 20 are represented numerically on the x axis. The relative width of the space allotted for each chromosome reflects the number of microsatellite markers typed for that chromosome. The y axis represents the LOD score. Two horizontal dashed lines denote genomewide thresholds for suggestive (P=0.63) and significant (P=0.05) linkage. The top panel shows a genomewide scan for atherosclerotic lesions using the nonparametric mode, and the bottom panel shows an autosome scan for atherosclerotic lesions using the parametric mode. For the latter scan, the X chromosome was not included due to a strong biased influence from the chromosome. QTL indicates quantitative trait locus; LOD, logarithm of odds.