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. 2009 Nov 13;14(6a):1347–1357. doi: 10.1111/j.1582-4934.2009.00966.x

Fig 6.

Fig 6

(A) Western blot analysis of hTERT proteins. CaCo-2 cells were treated with 25 μM 15-deoxy prostaglandin J2 (15d-PG J2) alone or in presence of GW9662 (15d-PJ J2 + GW) (left side) or with 200 μM rosiglitazone alone (Rosi) or in presence of GW9662 (Rosi + GW) (right side). (B) Western blot analysis of c-Myc proteins. CaCo-2 cells were treated with 25 μM 15-deoxy prostaglandin J2 (15d-PG J2) alone or in presence of GW9662 (15d-PJ J2 + GW) (left side) or with 200 μM rosiglitazone alone (Rosi) or in presence of GW9662 (Rosi + GW) (right side). (C) Western blot analysis of Mad1 proteins. CaCo-2 cells were treated with 25 μM 15-deoxy prostaglandin J2 (15d-PG J2) alone or in presence of GW9662 (15d-PJ J2 + GW) (left side) or with 200 μM rosiglitazone alone (Rosi) or in presence of GW9662 (Rosi + GW) (right side). Cells were collected 48 hrs after the treatment. Equal protein loading was confirmed by exposure of the membranes to the anti-β-actin antibody. Graphics represent the relative quantification of protein products performed by densitometric scanning. Data were normalized by using the β-actin signal and expressed as arbitrary densitometric units. Values represent the means ± S.D. of three independent experiments. Variance analysis: **P < 0.01 versus control.