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. 2013 Oct 21;110(45):E4256–E4265. doi: 10.1073/pnas.1316894110

Fig. 1.

Fig. 1.

Nitrite generation by Mtb within primary human macrophages cultured in nonhypoxic levels of oxygen. (A) Accumulation of nitrite in supernatant of macrophages in monolayer culture in 10% oxygen infected with WT (Wt) Mtb (MOI: 5; 5 × 105 bacteria) for the indicated times. Box and whisker plots depict median and 5th-95th percentiles of results from six donors. (B) As in A, but macrophages were uninfected or infected with narG-deficient Mtb (NarG) or WT or complemented (NarGc) strains. Results for uninfected macrophages and those infected with NarG Mtb overlap near zero. Means ± SD from one experiment representative of >10. (C) Lack of accumulation of nitrite in nitrate-depleted medium. Macrophages were infected in the presence (*) or absence of 5 mM nitrate. Means ± SD for one experiment representative of two. (D) As in B, but with macrophages maintained under either 21% or 10% oxygen. Means ± SD for one experiment representative of two. (E) Impact of rifampicin on nitrite production. Where indicated, macrophages were infected with WT Mtb (MOI: 5) for 3 d in 10% oxygen in the presence or absence of rifampicin (1 μg/mL). Rifampicin was added 1 d before (D-1), 1 d after (D1), or 2 d after (D2) infection. Uninfected macrophages were treated with rifampicin for the duration of the experiment. Nitrite was measured on day 3 after infection. Means ± SD, n = 2 experiments. (F) As in B, but using ∼20-fold higher numbers of macrophages grown on Cytodex surface microcarrier beads in 10% oxygen and then infected with Mtb (MOI: 5; 107 bacteria). Means ± SD, n = 2 experiments.