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. 2013 Nov 5;1(1):552–565. doi: 10.1016/j.redox.2013.10.008

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© 2013 The Authors

This is an open-access article distributed under the terms of the Creative Commons Attribution-NonCommercial-No Derivative Works License, which permits non-commercial use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig. 2 — Pharmacological inhibition of autophagy enhances AA, BSO or CCl₄ induced ROS stress. E47 cells were treated with AA, BSO or CCl₄ in the presence or absence of 3-MA or rapamycin for 48 h. (A) Lipid peroxidation was determined by measuring the formation of TBARs products. (B) GSH levels were determined with GSH reductase. (&P<0.05 compared with control; ^⁎, #P<0.05 compared with AA, BSO or CCl₄ alone, N=3).