Figure 3. Effect of PI-103 and rapamycin on critical components of the PI3K/AKT/mTOR signaling pathway.
A: cells were treated with PI-103 (0.75 µM) or rapamycin (0.1 µM), collected, and then lysed. Fifty micrograms of each lysate were electrophoresed on SDS-PAGE gels. B: Treatment with PI-103 for 8 h induced a dose-dependent dephosphorylation of Akt on Ser473 and of 4E-BP1 in CEM-R cells treated for 8 h. In A and B one representative of three different experiments is shown