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Proposed role of redox-dependent cross talk between mitochondria and NADPH oxidases (NOXs) in vascular dysfunction in hypertension. CVD, cardiovascular disease; NO, nitirc oxide; ONOO−, peroxynitrite; eNOS, endothelial NO synthase; GPx, glutathione peroxidase; Trx2, thioredoxin 2; mCat, catalase targeted to mitochondria; mKATP, ATP-sensitive K+ channel; ΔΨm, mitochondrial transmembrane potential.
