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. 2013 Sep 19;115(10):1481–1486. doi: 10.1152/japplphysiol.00745.2013

Table 1.

Action potential width at half-maximum height for control mice and those killed 1 h after exposure to 790-kPa air pressure for 2 h

Control Decompression
No manipulations 1.00 ± 0.04 (20) 1.45 ± 0.15 (9)*
    + PEG 0.92 ± 0.03 (3) 1.00 ± 0.03 (9)
Thrombocytopenia 1.05 ± 0.09 (3) 1.13 ± 0.05 (3)
    + 1400 W 1.11 ± 0.02 (3) 1.18 ± 0.04 (3)
    MPO-KO 1.14 ± 0.05 (6) 1.20 ± 0.07 (3)
    iNOS-KO 1.02 ± 0.06 (3) 1.18 ± 0.04 (3)

Values were normalized to the mean wild-type mouse control action potential width (3.97 ± 0.14 ms) and are displayed as means ± SE (n = individual mice studied).Where shown wild-type mice were first rendered thrombocytopenic, injected with 25 mg/kg 1400 W immediately before pressure exposure or injected with PEG telomere B (PEG) after decompression and killed 1 h later. Knock-out (KO) mice used in some studies lacked myeloperoxidase (MPO) or iNOS.

*

P < 0.05.