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. Author manuscript; available in PMC: 2015 Jan 1.
Published in final edited form as: J Allergy Clin Immunol. 2013 May 29;133(1):10.1016/j.jaci.2013.03.049. doi: 10.1016/j.jaci.2013.03.049

Fig 3.

Fig 3

CD11a polymorphisms influence allergic airway disease. A, Respiratory system resistance (RRS) values comparing BALB/c and BALB/cC57 congenic mice challenged with PBS or A niger conidia (AN). *P < .001 (n = 3 for BALB/c mice; n = 5 for all other groups). B, Total lung IFN-γ– and IL-4–producing cells. *P < .001. C, Respiratory system resistance values comparing C57BL/6 and C57Balb congenic mice. *P < .05, (n = 5). D, Total lung IFN-γ– and IL-4–producing cells. *P < .001. E and F, IL-5 and IL-13 levels from lung homogenates. *P < .05 (n = 5). Ach, Acetylcholine; ND, not detectable.