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. 2013 Nov 21;4(9):727–735. doi: 10.7150/jca.7576

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© Ivyspring International Publisher. This is an open-access article distributed under the terms of the Creative Commons License (http://creativecommons.org/licenses/by-nc-nd/3.0/). Reproduction is permitted for personal, noncommercial use, provided that the article is in whole, unmodified, and properly cited.

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Fig 5 — To explicit SRPK1 can increase DDP resistance of glioma cells in molecular levels. (A) The relative expression of SRPK1 and SRSF1 mRNAs in U251 cells treated by DDP or TMZ compared with control group in normoxic condition (*P<0.05). (B) RT-PCR detected SRPK1 and SRSF1 of U251 cells treated by DDP or TMZ compared with control group in hypoxic condition (*P<0.05, ** P<0.01). (C) The picture showed the expression of SRPK1 and SRSF1 in U251 cells by Western blot after 24h transfected with siRNA and treated or not with TMZ or DDP, β-Actin was used as an internal loading control.