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. 2013 Oct 1;27(19):2065–2071. doi: 10.1101/gad.228122.113

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© 2013 White; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genesdev.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 3.0 Unported), as described at http://creativecommons.org/licenses/by-nc/3.0/.

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Figure 1. — Fueling Ras. Ras-driven cancer cells alter glucose and glutamine metabolism and activate scavenging and self-cannibalization to supply metabolic pathways. Red indicates processes modulated by or required for Ras. (HBS) Hexosamine biosynthesis pathway; (ME1) malic enzyme 1; (MDH1) malate dehydrogenase; (GOT1) aspartate/glutamate–oxaloacetate transaminase 1; (OAA) oxaloacetate; (GLS1) glutaminase; (GLU) glutamate; (SBS) serine biosynthesis; (ROS) reactive oxygen species; (NABS) nucleic acid biosynthesis; (FAO) fatty acid oxidation.