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. Author manuscript; available in PMC: 2013 Dec 9.
Published in final edited form as: Am J Hypertens. 2008 Apr 24;21(7):10.1038/ajh.2008.178. doi: 10.1038/ajh.2008.178

Table 4.

Tracking (rTR), genetic (rG), and environmental (rE) correlations between visit 1 and 2

Measures rTR (%G) rG (95% CI) rE (95% CI)
IVSD 0.31 (49%) 0.37(0.13-0.59) 0.26(0.12-0.40)
LVPWD 0.29 (62%) 0.42(0.21-0.63) 0.19(0.06-0.33)
LVIDD 0.60 (58%) 1.00 0.38(0.25-0.49)
RWT 0.35 (54%) 0.48(0.25-0.69) 0.27(0.13-0.40)
LVM
 EA 0.49 (70%) 0.59(0.43-0.73) 0.35(0.18-0.50)
 AA, male 0.45 (64%) 0.44(0.17-0.66) 0.47(0.17-0.68)
 AA, female 0.53 (100%) 1.00 *
LVMI
 EA 0.50 (62%) 0.63(0.43-0.81) 0.37(0.21-0.52)
 AA, male 0.39 (52%) 0.34(0.02-0.61) 0.47(0.17-0.68)
 AA, female 0.48 (100%) 1.00 *

The correlations have been adjusted for the effects of age, gender, ethnicity, their interactions, and BMI.

%G, percentage of genetic contribution to the phenotypic tracking correlation. * = dismissed from the final model.

IVSD; interventricular septum in diastole; LVPWD; left ventricular posterior wall in diastole; LVIDD, left ventricular internal diameter in diastole; RWT, relative wall thickness; LVMI, left ventricular mass index (LVM/height 27)