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. 2013 Dec 10;7:251. doi: 10.3389/fncel.2013.00251

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Copyright © 2013 Gosselin, Meylan and Decosterd.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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(A–D) subcellular fractionation presented as bar histograms, showing the distribution patterns of glutamate and GABA transporters as a percentage over total signal in all fractions. A leftward shift is observed for EAAT-1 (A) and EAAT-2 (B). *p < 0.05; **p < 0.01, sham vs. SNI, t-test with Bonferroni correction for multiple comparisons (n = 5–6 rats for each condition). Arrows in (A) indicate signal detected in SNI samples with no counterparts in sham fractions. (E) Representative immunoblots of subcellular fractionation illustrating the shift of EAAT-1 and EAAT-2 content in SNI spinal cord, in the caveolin-1 enriched fractions. Arrowheads indicate fractions that are sites or EAAT-1 increase in SNI animals.