Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2013 Nov 7;1(6):e00143. doi: 10.1002/phy2.143

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2013 The Authors. Physiological Reports published by Wiley Periodicals, Inc. on behalf of the American Physiological Society and The Physiological Society

Re-use of this article is permitted in accordance with the Creative Commons Deed, Attribution 2.5, which does not permit commercial exploitation.

PMC Copyright notice

Histological analysis of E16.5 fetal Akt1^+/+, Akt1^+/−, and Akt1^−/− hearts, coronary blood vessels and myocardial capillaries. (A) Some Akt1^−/− fetal hearts displayed ventricular septum defects (VSDs; large arrow). Coronary vessels (>90 μm) located beneath the epicardium were rarely present in Akt1^−/− fetal hearts (small arrows). (B) Akt1^−/− myocardial veins are smaller and have a thinner perivascular wall than Akt1^+/+ veins (C) Akt1^−/− myocardium exhibited less myocardial capillaries than Akt1^+/+ myocardium. (D) Quantification of the amount of coronary vessels (>90 μm) per section. (E) Quantification of the amount of myocardial capillaries per high power field (HPF). (*P < 0.05).