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. Author manuscript; available in PMC: 2014 Dec 1.
Published in final edited form as: Epilepsy Behav. 2013 Oct 28;29(3):10.1016/j.yebeh.2013.09.039. doi: 10.1016/j.yebeh.2013.09.039

Table 1.

Demographic, clinical, and volumetric information of the participants

Healthy Control
(n = 20)
LTLE (n = 11) RTLE (n = 8)
Age 42.26 (15.80) 39.93 (11.54) 33.00 (14.56)
Education 15.85 (2.25) 14.27 (1.74) 14.00 (2.73)
BDI-II 5.1 (6.05)
Range: 0 – 21
10.82 (7.36)
Range: 1 – 21
9.86 (7.71)
Range: 0 – 18
Age of Onset - 17.50 (15.17) 20.00 (11.24)
Disease Duration - 21.86 (17.01) 12.75 (13.60)
Left Hippocampus Volume 3947.10 (368.88) 3359.09 (789.98)* 3975.25 (611.36)
Right Hippocampus Volume 3987.40 (366.37) 3980.55 (335.42) 3418.38 (1183.00)
Left Amygdala Volume 1526.25 (247.17) 1443.27 (358.29) 1669.88 (256.47)
Right Amygdala Volume 1720.15 (247.63) 1774.36 (266.75) 1743.50 (488.20)
Hippocampal Asymmetry −.01 (.05) −.18 (.19)*^ .20 (.26)^
Amygdala Asymmetry −.12 (.13) −.22 (. 17)^ −.02 (.20)^

Note:

*

denotes a patient group showing statistically significant difference from the control group at alpha of .05.

^

denotes LTLE and RTLE significantly different from each other at alpha of .05 Although the RTLE group show larger left hippocampal volume than the healthy control, the difference did not reach statistical significance, and by removing one outlier from RTLE group, their left hippocampal volume is smaller than healthy controls.