. Author manuscript; available in PMC: 2014 Sep 1.

Published in final edited form as: Obesity (Silver Spring). 2013 Aug 13;22(3):875–881. doi: 10.1002/oby.20517

Table 3.

Genetic correlations (ρ_G ± SE) and sample size between BMI measured at different times in the lifespan.

	Birth	Infancy	Childhood	Adolescence	Adult (19 yrs)	Adult (30 yrs)
Infancy (9 mos)	0.81 ± 0.29^a (340)
Childhood (7 yrs)	0.49 ± 0.15^b (485)	0.59 ± 0.09^b (488)
Adolescence (11 yrs)	0.20 ± 0.15^NS (479)	0.36 ± 0.12^b ^c (467)	0.96 ± 0.02^b (715)
Young adulthood (19 yrs)	0.19 ± 0.18^NS (410)	0.25 ± 0.13^NS (400)	0.80 ± 0.06^b (593)	0.84 ± 0.05^b (611)
Young adulthood (30 yrs)	0.68 ± 0.32^a ^c (290)	0.04 ± 0.18^NS (321)	0.62 ± 0.09^b (474)	0.78 ± 0.08^b (472)	0.99 ± 0.05^a (452)
Mid-Adulthood (40 yrs)	0.45 ± 0.30^NS (209)	−0.07 ± 0.20^NS (308)	0.34 ± 0.15^b ^c (385)	0.49 ± 0.14^b ^c (379)	0.64 ± 0.12^b (359)	0.96 ± 0.04^a (441)

^NS

Not significantly different from zero, indicating that all genes influencing BMI at one age are wholly different from those influencing BMI at the other age (i.e., there are gene-by-age effects)

Not significantly different from one, but significantly different from zero, indicating that all of the genes influencing BMI at one age are the same genes influencing BMI at another age.

Significantly different from zero, and significantly different from one, indicating that some by not all the genes influencing BMI at one age are the same genes influencing BMI at another age (i.e., there are gene-by-age effects)

Not significantly different from zero, after adjusting using Holm-Bonferroni.