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. Author manuscript; available in PMC: 2014 Jan 8.
Published in final edited form as: Atherosclerosis. 2011 Jun 25;219(1):10.1016/j.atherosclerosis.2011.06.038. doi: 10.1016/j.atherosclerosis.2011.06.038

Fig. 4.

Fig. 4

Immunohistochemistry of NT and eNOS in proximal aorta from Group 1, Group 3, and Group 4. There was no positive NT staining in Group 1. HNGF6A treatment to ApoE-deficient with HC (Group 4) showed weaker NT staining in the plaques than ApoE-deficient with HC (Group 3). (A) Bar=100µm. *P <0.05. eNOS was expressed in endothelial layer of Group 1, Group 3, and Group 4. eNOS staining in ApoE-deficient with HC (Group 3) was weaker than C57BL/6 with normal diet (Group 1). HNGF6A treatment to ApoE-deficient with HC (Group 4) restored the eNOS staining. (B) Bar = 200 µm. #P < 0.01 between 3 groups.