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. Author manuscript; available in PMC: 2014 Dec 6.
Published in final edited form as: Biochem Biophys Res Commun. 2013 Nov 15;442(0):10.1016/j.bbrc.2013.11.028. doi: 10.1016/j.bbrc.2013.11.028

Fig. 3.

Fig. 3

Increased K. pneumoniae infection and injury in spleens of GSNOR−/− mice. (A) Ratios of bacterial loads in the spleen (CFU/g) versus those in the lung (CFU/left lung) in wild-type (open circles, n = 5) and GSNOR−/− (closed diamonds, n = 4) mice. The ratios vary little within and between groups. (B) Representative spleen histology in K. pneumoniae-infected wild-type and GSNOR−/− mice. Bottom panels represent an expanded portion of the above sections outlined. Bar = 50 μm. (C) Histological scoring of infection-associated spleen injury revealed significant differences in wild-type (open; n = 11) and GSNOR−/− (closed; n = 10) mice. *, P < 0.03.