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OASIS, CREB4 and Luman N-termini form heterodimers to regulate Gcm1 expression. OASIS-CREB4 heterodimers activate transcription of Gcm1 during astrocyte differentiation. Conversely, the Luman N-terminus inhibits the formation of OASIS-CREB4 heterodimers by binding to the OASIS N-terminus, down-regulating transcription of Gcm1
