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Neonatal hyperoxia enhances MCP-1 expression in mice infected with influenza A virus. Adult Mcp-1 WT and KO mice exposed to RA or O2 at birth were intranasally infected with a sublethal dose of influenza A virus. Levels of MCP-1 in bronchoalveolar lavage fluid were determined on postinfection days 3, 5, and 7 (ND = not detected, n = 4–6 mice/group, *P < 0.05).
