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. Author manuscript; available in PMC: 2014 Feb 1.
Published in final edited form as: Acta Physiol (Oxf). 2012 Nov 26;207(2):358–368. doi: 10.1111/apha.12025

Figure 3.

Figure 3

A: Angiotensin (Ang)II-mediated vasoconstriction in aortas from SS-13BN rats on normal diet (untreated N=12, +L-NAME N=6) or hyper-caloric diet (untreated N=10, +L-NAME N=5). B: AngII vasoconstriction in aortas from SS rats on normal diet (untreated N=8, +L-NAME N=6) or hyper-caloric diet (untreated N=14, +L-NAME N=11). *P<0.05 for Emax in L-NAME-treated vs. untreated aortic segments from SS-13BN rats on normal diet. †P<0.05 for Emax in L-NAME-treated vs. untreated aortic segments from SS rats on hyper-caloric diet.