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. 2014 Jan 17;8:165–173. doi: 10.2147/DDDT.S51559

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© 2014 Gan et al. This work is published by Dove Medical Press Limited, and licensed under Creative Commons Attribution – Non Commercial (unported, v3.0) License

The full terms of the License are available at http://creativecommons.org/licenses/by-nc/3.0/. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed.

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Effect of 6-OHDA on total GluN1/GluN2A/GluN2B proteins of rat striatum.

Notes: (A and B) In the 6-OHDA–lesioned side, there was no difference in total GluN1 and GluN2A expression relative to other sides. (C) Total GluN2B levels in the 6-OHDA–lesioned sides were significantly higher than other three sides. The data are expressed in terms of means ± SEM and expressed as a percentage of the sham unlesioned side (n=3–4 per group). *P<0.05 compared with the sham unlesioned side.

Abbreviations: 6-OHDA, 6-hydroxydopamine; PD, Parkinson’s disease; U, unlesioned side; L, lesioned side; SEM, standard error of the mean.