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. 2013 Oct 24;42(2):1016–1025. doi: 10.1093/nar/gkt951

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© The Author(s) 2013. Published by Oxford University Press.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 3. — Subtelomeric and heterochromatin patterns. (A and B) Normalized levels of H2AX and related molecules as a function of the distance to (A) chromosome ends and (B) H3K9me3 domains. To adjust for possible bias in the density of nucleosomes themselves, mononucleosome sequencing data in active T cells (21) were used to estimate the incorporation rate of H2AX, γH2AX and H2AZ. (C) The number of single nucleotide variations occurred in cancer (14) was counted in 10-kb windows and averaged across H3K9me3 domains.