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. Author manuscript; available in PMC: 2014 Jan 27.
Published in final edited form as: Cancer Res. 2011 Jul 12;71(14):4846–4856. doi: 10.1158/0008-5472.CAN-11-0414

Figure 5.

Figure 5

GCNT2 is actively involved in EMT. A, the effect of GCNT2 on the EMT program. Left panels show the effect of knockdown of GCNT2 expression on the EMT program in 4T1 cells. Right panels show the effect of ectopic expression of GCNT2 on the EMT program in HMLE cells. B, representative pictures show morphologic change after knockdown of GCNT2 in 4T1 cells. C, the expression of E-cadherin at the RNA level by knockdown of GCNT2 in 4T1 cells (left) and ectopic expression of GCNT2 in HMLE cells (right). The Snail gene is used as a control. D, the effect of lactacystin on E-cadherin protein level in cells with and without GCNT2 expression.