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. Author manuscript; available in PMC: 2015 Feb 1.
Published in final edited form as: J Immunol. 2014 Feb 1;192(3):851–857. doi: 10.4049/jimmunol.1302831

Table 1.

Cell Type LPA receptors implicated Summary of findings LPA species (concentration) References

Natural Killer LPA1,2,3 Chemotaxis; mobilization of Ca2+ in activated cells; enhanced IFN-γ secretion; LPAR detected by flow cytometry 18:1 (1–10 μM) (38)
LPA2 Inhibited release of perforin and cytotoxic activity; enhances cAMP levels and activates PKA 14:0 (0.02–20 μM) (39)

Mast cells LPA1,2,3,4 (mRNA) Growth factor (human); induces proliferation and differentiation 18:1 (5 μM) (40)
NA Activates mast cells; causes tryptase release and vascular leakage in a mast-cell dependent manner in mice LPA gel (41)
LPA1,3 Histamine release, inhibited by DGPP 18:1 (10 μg/ml) (42)
LPA2 Production of MIP-1β, IL-8, MCP-1 in an IL-4 dependent manner 18:1 and 18:2 (1–50 μM) (43)
LPA5 (mRNA) MIP-1β release (human) 18:1 (1–5 μM) (44)

Neutrophils NA Infiltration of neutrophils in BALF of guinea pigs in Rho/ROCK dependent manner 18:1 (1–10 μg/ml) (45)
LPA1, 2 (mRNA and protein) Chemotaxis, greater in for neutrophils from pneumonia patients LPA (0.1–1 μM) (46)
NA Degranulation, PA production 18:1 (10–40 μM) (47)

Macrophage/Monocyte NA Survival factor for murine macrophages via PI3K 18:1 (7.7 μM) (48)
NA Upregulated IL-1β in mouse and human macrophages NA (49)
NA Monocyte migration to MCP-1 via iPLA2 at the leading edge NA (50)

B cells LPA2 (mRNA) B lymphoblast growth factor; MAPK activation; immunoglobulin production 18:1 (0.1–1 μM) (51)

Lymphatic endothelium LPA2 Proliferation and lymphangiogenesis via IL-8 18:1 (0.5–10 μM) (52)

Notes: NA = not available; BALF = bronchoalveolar lavage fluids; PA = phosphatidic acid