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. Author manuscript; available in PMC: 2014 Feb 3.
Published in final edited form as: J Biol Chem. 2007 May 1;282(25):18100–18107. doi: 10.1074/jbc.M702906200

FIGURE 3. Footshock alone is not sufficient to increase GluR1 phosphorylation, and the increase in GluR1 Ser-831 phosphorylation is NMDA receptor-dependent.

FIGURE 3

A and B, rats immediately shocked and removed (Shock/Remove, n = 8) showed no change in Ser-831 or Ser-845 phosphorylation compared with similarly handled, unshocked controls (Sham Control, n = 8) measured 1 h after training. C and D, rats trained after latent inhibition (Latent Inhibition, n = 8) showed no change in Ser-831 or Ser-845 phosphorylation compared with similarly treated, unshocked controls (Sham Control, n = 8) measured 1 h after training. E and F, rats trained in the presence of 0.4 mg/kg MK-801 (MK-801 Trained, n = 8) showed no change in Ser-831 or Ser-845 phosphorylation compared with similarly treated, unshocked controls (MK-801 Sham, n = 8) measured 1 h after training.