Skip to main content
. Author manuscript; available in PMC: 2014 Feb 4.
Published in final edited form as: J Med Genet. 2010 Nov 10;48(2):105–116. doi: 10.1136/jmg.2010.082552

Table 3.

Genotypes and phenotypes of 38 patients (30 families) with mutations in NPHP4, IQCB1, CEP290, RPGRIP1L, TMEM67, AHI1, CC2D2A and TTC21B and respective MPR statistics

Patient Diagnosis Kidney
ESRD
(yrs)
Eye Brain Liver Other Origin Gene Nucleotide Change(a)
(Zygosity State)
Amino Acid
Change
(Segregation)
Mutant Allele
Frequency
Mutation
Assignment(b)
Ref.
F962-23 NPHP NPHP—
(12)
- - - - Germany NPHP4 c.3364A>C (h) p.T1122P (p) 90/6,163 (1.5%) CEL I novel
c.305delA (h) p.N102fsX76 (m) 14/684 (2.0%) CEL I novel
F698 −21: NPHP NPHP (21) - - - - Belgium NPHP4 c.3068G>A (h) p.W1023X (m) 43/1,694 (2.5%) CEL I novel
−22: NPHP NPHP (11) - - - - c.3403delC (h) p.R1135fsX10 56/7,148 (0.8%) CEL I novel
A394 −21: SLS - RD, Nys - - - Germany IQCB1 c.1090C>T (h) p.R364X 77/3,945 (2.0%) CEL I neg. novel
−22: SLS NPHP (10) LCA, Nys - ? ? ? Seq 15 exons ?
F15 −21: SLS NPHP (11) LCA, Nys Atx, MR - - France CEP290 c.5649-50insA (h) p.L1884fsX23 (p) NA Seq 53 exons [33]
−24: SLS NPHP (15) LCA Atx, MR - - c.5850delT (h) p.F1950fsX15 (m) 34/4,784 (0.7%) CEL I [34]
F335 −22: SLS NPHP (13) LCA - - - France CEP290 c.2915T>C (H) p.L972P (p, m) 63/2,404 (2.6%) CEL I neg. novel
−25: SLS NPHP (>3) LCA - - -
F394-27 JBTS NPHP
(4)
RD, Nys,
Col
CVA,
Atx, MR
- MicCep Turkey CEP290 c.5668G>T (H) p.G1890X (*, m) 67/3,316 (2.0%) CEL I [10]
A1188-21 JBTS NPHP
(12)
- CVA, MR - - Pakistani CEP290 c.5668G>T (H) p.G1890X 36/1,028 (3.5%) CEL I [10]
A2029-21 JBTS NPHP
(2)
LCA MTS, MR HF - USA CEP290 c.5668G>T (h) p.G1890X 92/5460 (1.7%) Dir. seq. [10]
c.1189G>A (h) p.G397S 33/3408 (1.0%) CEL I novel
A1388-21 JBTS NPHP
(8)
Nys CVA, MR - deafness Germany CEP290 c.5649-50insA (h) p.L1884fsX23 NA Seq 53 exons [33]
c.136G>T (h) p.E46X 99/3,565 (2.8%) Dir. Seq novel
A1713-21 −21: JBTS NPHP LCA CVH - died 3 yrs Germany CEP290 c.6277delG (h) p.V2093fsX4 9/373 (2.4%) CEL I neg. [35]
−22: JBTS NPHP LCA CVH - - c.5182G>T (h) p.E1728X 5/229 (2.2%) Dir. Seq novel
A1715-21 JBTS NPHP
(10)
RD MR, Oph - CFA India CEP290 c.5445-8delAACT (h) p.L1815fsX4 (p) NA Seq 53 exons novel
c.5311G>T (h) p.E1771X (m) 9/370 (2.4%) CEL I neg. novel
A2420 −21: MKS MDK - OEC HDD - UK CEP290 c.1451delA (h) p.K484fsX8 33/4,111 (0.8%) CEL I novel
−22: MKS MDK - OEC HDD - ? ? ? Seq 18 exons ?
A2424 −21: MKS MDK - OEC,
DWM
- PD UK RPGRIP1L c.1829A>C (h) p.H610P 83/4,463 (1.9%) CEL I novel
−22: MKS MDK - OEC,
DWM
- PD c.721-4delAATG (h) p.N241fsX25 NA Seq 26 exons novel
F90-21 JBTS NPHP
(4)
Nys, RD CVA,
MR, Atx,
HF PD,
MicCep
Germany TMEM67 c.1843T>C (h) p.C615R 39/843 (4.6%) CEL I neg. [36]
c.755T>C (h) p.M252T (p) 36/1,243 (2.9%) CEL I [37]
F96-22 JBTS NPHP
(22)
Col, RD CVA,
MR, Atx
- - Germany TMEM67 c.755T>C (h) p.M252T (m) 42/1,538 (2.7%) CEL I [37]
c.2498T>C (h) p.I833T (p) 165/13,292
(1.2%)
CEL I [38]
F278-21 JBTS NPHP
(14)
- Atx, MR HF,
CP
- Germany TMEM67 c.1843T>C (h) p.C615R 234/6,373 (3.7%) CEL I neg. [36]
c.755T>C (h) p.M252T 13/1,409 (0.9%) CEL I [37]
F315-21 JBTS NPHP
(9)
Nys CVH, MR HF - Germany TMEM67 c.1843T>C (h) p.C615R 234/6,373 (3.7%) CEL I neg. [36]
c.1911C>A (h) p.F637L 17/1,115 (1.5%) CEL I novel
F480-22 JBTS NPHP
(0.2)
- CVH - - Germany TMEM67 c.1387C>T (h) p.R463X (m) 7/320 (2.2%) Dir. Seq novel
c.2891C>T (h) p.T964I (p) 10/528 (1.9%) CEL I novel
F459-22 JBTS NPHP
(30)
Col CVH,
ATX, MR
HF,
BDP
hearing
loss
USA TMEM67 c.986A>C (h)
c.2556+1G>A (h)
p.K329T (p)
splice
95/3,313 (2.9%)
172/10,393
(1.7%)
CEL I
CEL I
novel
novel
F631-21 JBTS NPHP
(21)
Col MR, HF - Germany TMEM67 c.1045T>C (h) p.L349S NA Seq 28 exons [37]
c.1843T>C (h) p.C615R (p) 39/843 4.6%) CEL I neg. [36]
A77-21 JBTS NPHP
(7)
OA CVH,
MR
HF aortic
stenosis
Germany TMEM67 c.622A>T (h)
c.2168A>G (h)
p.R208X
p.Y723C (m)
123/6,069 (2.0%)
13/905 (1.4%)
CEL I
CEL I
[37]
novel
A971-21 JBTS NPHP
(7)
Nys CVH,
MR, Atx
HF,
CP
- Germany TMEM67 c.622A>T (h) p.R208X NA Seq 28 exons [37]
c.1843T>C (h) p.C615R 234/6,373 (3.7%) CEL I neg. [36]
A3208-21 JBTS NPHP
(28)
Col MTS, MR - epilepsy UK TMEM67 c.1351C>T (h) p.R451X 75/5,041 (1.5%) CEL I [39]
c.2018T>C (h) p.V673A 270/13,865
(1.9%)
CEL I novel
F787-21 JBTS - - CVH - - Germany AHI1 c.2671C>T (h) p.R891X 112/4,540 (2.5%) CEL I novel
? ? ? Seq 28 exons ?
F434-21 JBTS - OMAC CVH, HT,
Atx, MR
- - Germany CC2D2A c.517C>T (h) p.R173X (p) 63/3,771 (1.7%) Dir. Seq [40]-
c.1676T>C (h) p.L559P (m) 67/6,139 (1.1%) CEL I novel
A2421-21 MKS MDK - OEC HDD - UK CC2D2A c.3544T>C (h) p.W1182R 5/500 (1.0%) CEL I novel
c.3774_5insT (h) p.E1259fsX1 NA Seq 35 exons novel
A2426-21 MKS MDK - OEC - CFA,
OHy
UK CC2D2A c.685_7delGAA (h) p.E229del NA Seq 35 exons novel
c.3893T>A (h) p.V1298D 295/18,699
(1.6%)
CEL I novel
F244-25 infantile
NPHP
NPHP
(1.5)
- MR, Oph HF SI, VC,
Bro
Turkey TTC21B c.626C>T (h) p.P209L (m) 334/11,391
(2.9%)
Dir. Seq [25]
c.1654-7delTGTC (h) p.C552fsX1 (p) NA Seq 29 exons [25]
F514- −21: NPHP NPHP (7) - - PSC - Swiss TTC21B c.448T>C (h) p.W150R (m) 53/5,802 (0.9%) CEL I [25]
−22: NPHP NPHP(2) - - PSC SI, VUR c.3264-3C>G (h) splice site (p) NA Seq 29 exons [25]
A34-21 infantile
NPHP
NPHP
(2)
RD - HF SI, short
phalanges
Portuguese TTC21B c.2758-2A>G (h) splice site (m) 92/4,905 (1.9%) CEL I neg. [25]
c.626C>T (h) p.P209L (p) 334/11,391
(2.9%)
Dir. Seq [25]
a

All mutations were absent from at least 96 healthy control subjects. Mutation numbering is based on cDNA position in reference sequences indicated in Table 1 with +1 corresponding to the A of the ATG translation initiation codon.

b

The mutation carrier assignment was performed by heteroduplex based CEL I endonuclease screening (CEL I) or by direct Sanger sequencing of the respective 24 DNA samples (Dir. Seq). In cases CEL I analysis was inconclusive or negative (CEL I neg.) all 24 DNA samples from the respective pool were directly sequenced for one exon. In cases where only one mutated allele was found, all exons of the respective gene were sequenced and exon numbers are indicated (Seq # exons).

?

second mutation not detected;

*

no maternal or paternal DNA available; Atx, ataxia; BDP, bilary ductal proliferation; Bro, bronchiectasis; CFA, craniofacial abnormalities; CP, cholangio-dysplasia; CVA, cerebellar vermis aplasia; CVH, cerebellar vermis hypoplasia; DWM, Dandy walker malformation; ESRD, end-stage renal disease; (h), heterozygous; (H), homozygous; HDD, hepatic developmental defects; HF, hepatic fibrosis; HT, muscle hypotonia; LCA, Leber congenital amaurosis; (m), heterozygous mutation identified in mother; MDK, multicystic dysplastic kidneys; MicCep, microcephaly; MR, mental retardation; MTS, molar tooth sign; NA, no data available; Nys, nystagmus; OA, opticus atrophy; OEC, occipital encephalocele; OHy, oligohydramnios; OMAC,oculomotor apraxia type Cogan; Oph, ophistotonus; (p), heterozygous mutation identified in father; PD, polydactyly; PSC, primary sclerosing cholangitis; RD, retinal dystrophy; SI, situs inversus; VC, vitium cordis; VUR, vesicoureteral reflux.