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. Author manuscript; available in PMC: 2014 Feb 6.
Published in final edited form as: Am J Physiol Heart Circ Physiol. 2005 Sep 30;290(2):H732–H740. doi: 10.1152/ajpheart.00747.2005

Fig. 1.

Fig. 1

Amplification of total RNA from isolated microvessels by RT-PCR produced a 768-bp ICAM-1 cDNA fragment at 0 (control), 0.25, 0.50, 0.75, and 24 h postinduction of inflammatory pain. Control (0 h) showed a basal level of ICAM-1 mRNA. Remaining time points indicate an increase (although not quantitatively) in mRNA expression immediately after treatment (0.25, 0.50, and 0.75 h) and at 24 h. β-Actin was used as an internal control. (n = 3 per time point).