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. 2013 Nov 28;13(1):156–164. doi: 10.1111/acel.12165

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© 2013 The Authors. Aging Cell published by the Anatomical Society and John Wiley & Sons Ltd.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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ndg-4 and insulin-like signaling extend stress resistance and lifespan additively. (A) Thermotolerance of ndg-4(lb108) mutants is partially daf-16-dependent. (B) Lifespan extension of ndg-4(lb018) mutants is partially daf-16-dependent. (C) The lifespan extension of ndg-4(lb108) mutants is partially dependent on aak-2. (D) daf-2 knockdown by RNAi increases the lifespan of ndg-4 mutants by nearly 100%.