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. 2013 Dec 24;6(1):54–65. doi: 10.3390/toxins6010054

Figure 2.

Figure 2

Effect of penta-O-galloyl-β-d-glucose (PGG) on phosphatidylserine exposure (A) Confocal images of FITC-dependent fluorescence (upper panels) and light microscopy (lower panels) of human erthrocytes following exposure for 48 h to Ringer solution without (left panel) and with (right panel) the presence of 25 µM penta-O-galloyl-β-d-glucose; (B) Original histogram of annexin V binding of erythrocytes following exposure for 48 h to Ringer solution without (grey) and with (black) the presence of 25 µM penta-O-galloyl-β-d-glucose. (C) Arithmetic means ± SEM of erythrocyte annexin V binding (n = 8) following incubation for 48 h to Ringer solution without (white bar) or with (black bars) the presence of penta-O-galloyl-β-d-glucose (1–50 µM). * (p < 0.05), *** (p < 0.001) indicates significant difference from the absence of penta-O-galloyl-β-d-glucose (ANOVA).