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. Author manuscript; available in PMC: 2014 Feb 11.
Published in final edited form as: Biochem Biophys Res Commun. 2009 Nov 27;391(1):852–856. doi: 10.1016/j.bbrc.2009.11.151

Fig. 1.

Fig. 1

Effect of UCH-L1 over-expression on TNFα-induced VSMC proliferation. (A) TNFα-induced proliferation of rat aortic smooth muscle cells (RASMCs). Cell proliferation was assessed by measuring [3H]thymidine update as described in “Methods”. * p<0.05 vs TNFα (−), n=4. (B) Adenoviral over-expression of UCH-L1 in RASMCs. Results are representative of three independent Western blot analysis of UCH-L1 in RASMCs infected with or without adenovirus of UCH-L1. (C) Effect of over-expression of UCH-L1 on TNFα-induced RASMC proliferation. Cells infected with Ad-UCH-L1 or Ad-βGal were stimulated with or without TNFα (5 ng/ml) as indicated for 40 hours. * p<0.05 vs TNFα (−), n=4.