Table 1. Enhanced SIMR in rcd1 double mutants.

Double mutant	Biological process altered	Reference to double mutant	Reference to single mutant
rcd1 axr1	Subunit of RUB1 activating enzyme that regulates protein degradation activity of Skp1-Cullin-F-box complexes.	[14]	[120]
rcd1 noa1	Decreased nitric oxide production+required for chloroplast ribosome assembly	[121]	[122]
rcd1 noa2	Unknown	[121]	Not available
rcd1 sro1	Closest homolog to rcd1	[31], [35],	[31], [35],
rcd1 vtc1	Decreased ascorbic acid concentration+altered stress responses	Figure 7	[123]
rcd1 vtc2	Decreased ascorbic acid concentration+altered stress responses	Figure 7	[123]
rcd1 snc1	Immune response	[41]	[41]
rcd1 rbohF	ROS production	[41]	[124]
Other rcd1 double mutants in hormone signaling i.e., rcd1 ein2, rcd1 jar1, rcd1 coi1-16, rcd1 npr1 have the same growth phenotype as rcd1.		[10], [14], [28]

Enhanced SIMR, defined as increased growth defects and/or dwarfism, is observed in the listed rcd1 double mutants. The biological process(es) altered in the mutant crossed with rcd1 is briefly summarized. Of the double mutants used for cell death experiments (Figure 5 and 6) only rcd1 vtc2 displayed enhanced SIMR. In addition rcd1 agb1-2, rcd1 gpa1-4 and rcd1 agb1-2 gpa1-4 displayed leaf characteristics of both parents: round leaves like the G-protein mutants and wavy, bushy leaves like rcd1.