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. 2014 Mar 1;141(5):1036–1046. doi: 10.1242/dev.106534

Fig. 5.

Fig. 5.

Erm-dependent restriction of developmental potential in immature INPs leads to attenuated competence to respond to Dpn and E(spl)mγ in INPs. (A-E) Co-expression of erm can suppress the supernumerary neuroblast phenotype induced by mis-expression of dpn, as seen in B-E. Scale bar: 40 μm. (A) Quantification of total Wor+Ase- cells (including type II neuroblasts and Ase- immature INPs) per brain lobe of the indicated genotypes. (F) Removing dpn function suppresses supernumerary neuroblast formation in erm-null brains. Quantification of total Wor+Ase- cells (including type II neuroblasts and Ase- immature INPs) per clone for the indicated genotypes. (F) Third instar larval brains carrying GFP-marked mosaic clones derived from single neuroblasts of the indicated genotypes were stained for GFP, Wor, Ase, Pros and Elav. (G) Co-expression of erm can suppress the supernumerary neuroblast phenotype induced by mis-expression of E(spl)mγ. Quantification of total type II neuroblasts (Dpn+Ase-) per clone for the indicated genotypes.