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. Author manuscript; available in PMC: 2014 Nov 15.
Published in final edited form as: Dev Biol. 2013 Sep 25;383(2):214–226. doi: 10.1016/j.ydbio.2013.09.019

Table 1.

Transplantation experiments indicate a cell-autonomous role for tal1 during endocardial morphogenesis.

Transplant type Total endocardial contribution Atrial endocardial contribution Ventricular endocardial contribution Endothelial contribution Myocardial contribution
WT to WT 15/184 (8.2%) 10/15 (66.7%) 9/15 (60.0%) 134/184 (72.8%) 37/214 (17.3%)
tal1 MO to WT 21/299 (7.0%) 2/21 (9.5%)* 19/21 (90.5%) 103/299 (34.4%)* 52/451 (11.5%)
WT to tal1 MO 9/238 (3.8%) 8/9 (88.9%) 2/9 (22.2%) 185/238 (77.7%)

Each fraction reports the number of embryos in which donor cells contributed to a particular tissue divided by the total number of mosaic embryos scored. The fractions representing atrial and ventricular endocardial contributions indicate the number of embryos in which donor cells contributed to the atrial or ventricular endocardium divided by the total number of embryos displaying donor contribution to the endocardium. In some cases, donor cells contributed to both the ventricular and atrial endocardium (Fig. 2O). Asterisks indicate statistically significant differences from control transplants (wild-type into wild-type), as determined by Fisher’s exact test (p < .05).