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. Author manuscript; available in PMC: 2015 Feb 1.
Published in final edited form as: J Immunol. 2013 Dec 23;192(3):929–939. doi: 10.4049/jimmunol.1302171

Figure 6. EAE shifts from classic to atypical disease in B cell-deficient recipients of Th1 cells.

Figure 6

EAE was induced by adoptive transfer of Th1- or Th17-skewed cells into C3HeB/Fej WT or μMT recipients. The percentages of classic and atypical EAE observed in recipient mice that developed EAE are shown (n=7–24 per group). A significant difference between WT and μMT recipients of Th1 cells was observed; ****p < 0.0001, Fisher’s exact test.