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. 2014 Feb 24;15:155. doi: 10.1186/1471-2164-15-155

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Copyright © 2014 Luo et al.; licensee BioMed Central Ltd.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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GRO-seq and Pol II ChIP-seq reveal the dynamics of TNFα-dependent transcription in AC16 cells. GRO-seq and ChIP-seq in AC16 cells treated with a time course of TNFα reveals the dynamics of transcription. A) Metagene representations showing the average GRO-seq read distributions ± 4 kb around the TSSs of upregulated (top) or downregulated (bottom) RefSeq genes over a time course of TNFα treatment in AC16 cells. B and C) Metagene representations showing the average Pol II (B) and NF-κB p65 (C) ChIP-seq read distributions ± 4 kb around the TSSs of upregulated (left) or downregulated (right) RefSeq genes at 0 and 30 min of TNFα treatment.